Dr Chunling Huang

NHMRC Early Career Fellow
Northern Clinical School


Map

Selected grants

2019

  • A novel therapeutic target for diabetic nephropathy; Huang C; DVC Research/Laffan Fellowships.

2018

  • A novel and unique protein i-body for the treatment of chronic kidney disease through targeting CXCR4; Pollock C, Foley M, Chen X, Huang C; National Health and Medical Research Council (NHMRC)/Project Grants.

2016

  • Targeting microRNA-200b in diabetic nephropathy using two novel delivery systems; Huang C; National Health and Medical Research Council (NHMRC)/Early Career Fellowships.

2015

  • Thioredoxin interacting protein (Txnip): A novel regulator of tubular autophagy in diabetic nephropathy; Huang C; University of Sydney/Early Career Researcher.

Selected publications

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Journals

  • Yi, H., Huang, C., Shi, Y., Cao, Q., Zhao, Y., Zhang, L., Chen, J., Pollock, C., Chen, X. (2018). Metformin attenuates folic-acid induced renal fibrosis in mice. Journal of Cellular Physiology, 233(9), 7045-7054. [More Information]
  • Huang, C., Zhang, L., Shi, Y., Yi, H., Zhao, Y., Chen, J., Pollock, C., Chen, X. (2018). The KCa31 blocker TRAM34 reverses renal damage in a mouse model of established diabetic nephropathy. PloS One, 13(2), 1-15. [More Information]
  • Huang, C., Lin, Z., Cheng, D., Braet, F., Pollock, C., Chen, X. (2016). KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways. Scientific Reports, 6, 1-12. [More Information]
  • Peeters, M., Huang, C., Vonk, L., Lu, Z., Bank, R., Helder, M., Doulabi, B. (2016). Optimisation of high-quality total ribonucleic acid isolation from cartilaginous tissues for real-time polymerase chain reaction analysis. Bone & Joint Research, 5(11), 560-568. [More Information]
  • Huang, C., Zhang, Y., Kelly, D., Tan, C., Gill, A., Cheng, D., Braet, F., Park, J., Sue, C., Pollock, C., Chen, X. (2016). Thioredoxin interacting protein (TXNIP) regulates tubular autophagy and mitophagy in diabetic nephropathy through the mTOR signaling pathway. Scientific Reports, 6, 1-13. [More Information]
  • Huang, C., Pollock, C., Chen, X. (2015). KCa3.1: a new player in progressive kidney disease. Current Opinion In Nephrology And Hypertension, 24(1), 61-66. [More Information]
  • Huang, C., Pollock, C., Chen, X. (2014). High Glucose Induces CCL20 in Proximal Tubular Cells via Activation of the KCa3.1 Channel. PloS One, 9(4), 1-8. [More Information]
  • Huang, C., Day, M., Poronnik, P., Pollock, C., Chen, X. (2014). Inhibition of KCa3.1 suppresses TGF-Beta1 induced MCP-1 expression in human proximal tubular cells through Smad3, p38 and ERK1/2 signaling pathways. The International Journal of Biochemistry and Cell Biology, 47(1), 1-10. [More Information]
  • Gangadharan Komala, M., Gross, S., Mudaliar, H., Huang, C., Pegg, K., Mather, A., Shen, S., Pollock, C., Panchapakesan, U. (2014). Inhibition of Kidney Proximal Tubular Glucose Reabsorption Does Not Prevent against Diabetic Nephropathy in Type 1 Diabetic eNOS Knockout Mice. PloS One, 9(11), 1-12. [More Information]
  • Huang, C., Shen, S., Ma, Q., Gill, A., Pollock, C., Chen, X. (2014). KCa3.1 mediates activation of fibroblasts in diabetic renal interstitial fibrosis. Nephrology Dialysis Transplantation, 29(2), 313-324. [More Information]
  • Huang, C., Pollock, C., Chen, X. (2014). Role of the potassium channel KCa3.1 in diabetic nephropathy. Clinical Science, 127(7), 423-433. [More Information]
  • Huang, C., Lin, M., Cheng, D., Braet, F., Pollock, C., Chen, X. (2014). Thioredoxin-interacting protein mediates dysfunction of tubular autophagy in diabetic kidneys through inhibiting autophagic flux. Laboratory Investigation, 94(3), 309-320. [More Information]
  • Huang, C., Shen, S., Ma, Q., Chen, J., Gill, A., Pollock, C., Chen, X. (2013). Blockade of KCa3.1 Ameliorates Renal Fibrosis Through the TGF-β1/Smad Pathway in Diabetic Mice. Diabetes, 62(8), 2923-2934. [More Information]

2018

  • Yi, H., Huang, C., Shi, Y., Cao, Q., Zhao, Y., Zhang, L., Chen, J., Pollock, C., Chen, X. (2018). Metformin attenuates folic-acid induced renal fibrosis in mice. Journal of Cellular Physiology, 233(9), 7045-7054. [More Information]
  • Huang, C., Zhang, L., Shi, Y., Yi, H., Zhao, Y., Chen, J., Pollock, C., Chen, X. (2018). The KCa31 blocker TRAM34 reverses renal damage in a mouse model of established diabetic nephropathy. PloS One, 13(2), 1-15. [More Information]

2016

  • Huang, C., Lin, Z., Cheng, D., Braet, F., Pollock, C., Chen, X. (2016). KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways. Scientific Reports, 6, 1-12. [More Information]
  • Peeters, M., Huang, C., Vonk, L., Lu, Z., Bank, R., Helder, M., Doulabi, B. (2016). Optimisation of high-quality total ribonucleic acid isolation from cartilaginous tissues for real-time polymerase chain reaction analysis. Bone & Joint Research, 5(11), 560-568. [More Information]
  • Huang, C., Zhang, Y., Kelly, D., Tan, C., Gill, A., Cheng, D., Braet, F., Park, J., Sue, C., Pollock, C., Chen, X. (2016). Thioredoxin interacting protein (TXNIP) regulates tubular autophagy and mitophagy in diabetic nephropathy through the mTOR signaling pathway. Scientific Reports, 6, 1-13. [More Information]

2015

  • Huang, C., Pollock, C., Chen, X. (2015). KCa3.1: a new player in progressive kidney disease. Current Opinion In Nephrology And Hypertension, 24(1), 61-66. [More Information]

2014

  • Huang, C., Pollock, C., Chen, X. (2014). High Glucose Induces CCL20 in Proximal Tubular Cells via Activation of the KCa3.1 Channel. PloS One, 9(4), 1-8. [More Information]
  • Huang, C., Day, M., Poronnik, P., Pollock, C., Chen, X. (2014). Inhibition of KCa3.1 suppresses TGF-Beta1 induced MCP-1 expression in human proximal tubular cells through Smad3, p38 and ERK1/2 signaling pathways. The International Journal of Biochemistry and Cell Biology, 47(1), 1-10. [More Information]
  • Gangadharan Komala, M., Gross, S., Mudaliar, H., Huang, C., Pegg, K., Mather, A., Shen, S., Pollock, C., Panchapakesan, U. (2014). Inhibition of Kidney Proximal Tubular Glucose Reabsorption Does Not Prevent against Diabetic Nephropathy in Type 1 Diabetic eNOS Knockout Mice. PloS One, 9(11), 1-12. [More Information]
  • Huang, C., Shen, S., Ma, Q., Gill, A., Pollock, C., Chen, X. (2014). KCa3.1 mediates activation of fibroblasts in diabetic renal interstitial fibrosis. Nephrology Dialysis Transplantation, 29(2), 313-324. [More Information]
  • Huang, C., Pollock, C., Chen, X. (2014). Role of the potassium channel KCa3.1 in diabetic nephropathy. Clinical Science, 127(7), 423-433. [More Information]
  • Huang, C., Lin, M., Cheng, D., Braet, F., Pollock, C., Chen, X. (2014). Thioredoxin-interacting protein mediates dysfunction of tubular autophagy in diabetic kidneys through inhibiting autophagic flux. Laboratory Investigation, 94(3), 309-320. [More Information]

2013

  • Huang, C., Shen, S., Ma, Q., Chen, J., Gill, A., Pollock, C., Chen, X. (2013). Blockade of KCa3.1 Ameliorates Renal Fibrosis Through the TGF-β1/Smad Pathway in Diabetic Mice. Diabetes, 62(8), 2923-2934. [More Information]

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