Dr Rose Boutros

Lecturer
Medicine, Children's Medical Research Institute


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Research interests

My research focuses on identifying the role of centrosome defects in tumourigenesis.

Current projects within my group include: - the role of protein phosphorylation in regulating centrosome duplication - the role of the centrosome in the response to DNA damage

Selected grants

2012

  • Role of CDK2 and PLK4 kinases in regulating centrosome duplication and their effects on tumourigenesis; Boutros R; Cure Cancer Australia Foundation/Research Support.

Selected publications

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Journals

  • Boutros, R., Mondesert, O., Lorenzo, C., Astuti, P., McArthur, G., Chircop (nee Fabbro), M., Ducommun, B., Gabrielli, B. (2013). CDC25B Overexpression Stabilises Centrin 2 and Promotes the Formation of Excess Centriolar Foci. PLoS One, 8(7), 1-13. [More Information]
  • Boutros, R., Lorenzo, C., Mondesert, O., Jauneau, A., Oakes, V., Dozier, C., Gabrielli, B., Ducommun, B. (2011). CDC25B associates with a centrin 2-containing complex and is involved in maintaining centrosome integrity. Biology of the Cell, 103(2, Suppl. 1), 55-68. [More Information]
  • Shehata, M., Bie'che, I., Boutros, R., Weidenhofer, J., Fanayan, S., Spalding, L., Zeps, N., Bright, R., Byth Wilson, K., Lidereau, R., Byrne, J. (2008). Nonredundant Functions for Tumor Protein D52-Like Proteins Support Specific Targeting of TPD52. Clinical Cancer Research, 14(16), 5050-5060. [More Information]
  • Boutros, R., Byrne, J. (2005). D53 (TPD52L1) is a cell cycle-regulated protein maximally expressed at the G2-M transition in breast cancer cells. Experimental Cell Research: emphasizing molecular approaches to cell biology, 310(1), 152-165. [More Information]
  • Shehata, M., Boutros, R., Bright, R., Byrne, J. (2005). In vitro models for tumor protein d52 function in cancer cells. Breast Cancer Research, 7(Suppl 2), S37-S37.
  • Boutros, R., Fanayan, S., Shehata, M., Byrne, J. (2004). The Tumor Protein D52 Family: Many Pieces, Many Puzzles. Biochemical and Biophysical Research Communications, 325(4), 1115-1121.
  • Boutros, R., Bailey, A., Wilson, S., Byrne, J. (2003). Alternative splicing as a mechanism for regulating 14-3-3 binding: interactions between hD53 (TPD52L1) and 14-3-3 proteins. Journal of Molecular Biology, 332(3), 675-687.

2013

  • Boutros, R., Mondesert, O., Lorenzo, C., Astuti, P., McArthur, G., Chircop (nee Fabbro), M., Ducommun, B., Gabrielli, B. (2013). CDC25B Overexpression Stabilises Centrin 2 and Promotes the Formation of Excess Centriolar Foci. PLoS One, 8(7), 1-13. [More Information]

2011

  • Boutros, R., Lorenzo, C., Mondesert, O., Jauneau, A., Oakes, V., Dozier, C., Gabrielli, B., Ducommun, B. (2011). CDC25B associates with a centrin 2-containing complex and is involved in maintaining centrosome integrity. Biology of the Cell, 103(2, Suppl. 1), 55-68. [More Information]

2008

  • Shehata, M., Bie'che, I., Boutros, R., Weidenhofer, J., Fanayan, S., Spalding, L., Zeps, N., Bright, R., Byth Wilson, K., Lidereau, R., Byrne, J. (2008). Nonredundant Functions for Tumor Protein D52-Like Proteins Support Specific Targeting of TPD52. Clinical Cancer Research, 14(16), 5050-5060. [More Information]

2005

  • Boutros, R., Byrne, J. (2005). D53 (TPD52L1) is a cell cycle-regulated protein maximally expressed at the G2-M transition in breast cancer cells. Experimental Cell Research: emphasizing molecular approaches to cell biology, 310(1), 152-165. [More Information]
  • Shehata, M., Boutros, R., Bright, R., Byrne, J. (2005). In vitro models for tumor protein d52 function in cancer cells. Breast Cancer Research, 7(Suppl 2), S37-S37.

2004

  • Boutros, R., Fanayan, S., Shehata, M., Byrne, J. (2004). The Tumor Protein D52 Family: Many Pieces, Many Puzzles. Biochemical and Biophysical Research Communications, 325(4), 1115-1121.

2003

  • Boutros, R., Bailey, A., Wilson, S., Byrne, J. (2003). Alternative splicing as a mechanism for regulating 14-3-3 binding: interactions between hD53 (TPD52L1) and 14-3-3 proteins. Journal of Molecular Biology, 332(3), 675-687.

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