Dr Wendy Gold

Senior Research Fellow
Sub-Dean (ECR)
Child & Adolescent Health, Children's Hospital, Westmead

Telephone +61 2 9845 1446

Website Honours in Medical Sciences at Wesmead

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Clinical Specialty

Paediatrics

Selected publications

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Journals

  • Zhang, J., Barbaro, P., Guo, Y., Alodaib, A., Li, J., Gold, W., Ades, L., Keating, B., Xu, X., Teo, J., Christodoulou, J., et al (2016). Utility of next-generation sequencing technologies for the efficient genetic resolution of haematological disorders. Clinical Genetics, 89(2), 163-172. [More Information]
  • Gold, W., Lacina, T., Cantrill, L., Christodoulou, J. (2015). MeCP2 deficiency is associated with reduced levels of tubulin acetylation and can be restored using HDAC6 inhibitors. Journal of Molecular Medicine, 93(1), 63-72. [More Information]
  • Gold, W., Christodoulou, J. (2015). The utility of next-generation sequencing in gene discovery for mutation-negative patients with Rett syndrome. Frontiers in Cellular Neuroscience, 9, 1-6. [More Information]
  • Foley, A., Menezes, M., Pandraud, A., Gonzalez, M., Al-Odaib, A., Abrams, A., Sugano, K., Yonezawa, A., Manzur, A., Burns, J., Wang, M., Carpenter, K., Lek, M., Gold, W., Ouvrier, R., Christodoulou, J., et al (2014). Treatable childhood neuronopathy caused by mutations in riboflavin transporter RFVT2. Brain, 137(Pt 1), 44-56. [More Information]

2016

  • Zhang, J., Barbaro, P., Guo, Y., Alodaib, A., Li, J., Gold, W., Ades, L., Keating, B., Xu, X., Teo, J., Christodoulou, J., et al (2016). Utility of next-generation sequencing technologies for the efficient genetic resolution of haematological disorders. Clinical Genetics, 89(2), 163-172. [More Information]

2015

  • Gold, W., Lacina, T., Cantrill, L., Christodoulou, J. (2015). MeCP2 deficiency is associated with reduced levels of tubulin acetylation and can be restored using HDAC6 inhibitors. Journal of Molecular Medicine, 93(1), 63-72. [More Information]
  • Gold, W., Christodoulou, J. (2015). The utility of next-generation sequencing in gene discovery for mutation-negative patients with Rett syndrome. Frontiers in Cellular Neuroscience, 9, 1-6. [More Information]

2014

  • Foley, A., Menezes, M., Pandraud, A., Gonzalez, M., Al-Odaib, A., Abrams, A., Sugano, K., Yonezawa, A., Manzur, A., Burns, J., Wang, M., Carpenter, K., Lek, M., Gold, W., Ouvrier, R., Christodoulou, J., et al (2014). Treatable childhood neuronopathy caused by mutations in riboflavin transporter RFVT2. Brain, 137(Pt 1), 44-56. [More Information]

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